Why the study?
Does myocardial infarction alter the electrophysiological properties and stretch sensitivity of atrial fibroblasts, contributing to bradyarrhythmia?
Does myocardial infarction alter the electrophysiological properties and stretch sensitivity of atrial fibroblasts, contributing to bradyarrhythmia?
Changes in the susceptibility of atrial fibroblasts to mechanical stretch may contribute to bradyarrhythmia during postinfarct remodeling.
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May implicate atrial fibroblasts in post-MI bradyarrhythmia; hypothesis-generating and leaves human translation open.
Kamkin et al. (2002) studied this question.
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