Key result
Huo-Xue-Xiao-Zhong Decoction reduces thrombus burden and endothelial dysfunction by downregulating SOAT2 and suppressing AKT-ERK.
Why the study?
Huo-Xue-Xiao-Zhong Decoction prevents deep vein thrombosis in clinical trials, but the underlying molecular mechanisms remain to be elucidated.
Does Huo-Xue-Xiao-Zhong Decoction prevent thrombus formation in preclinical models of deep vein thrombosis?
Population
Mouse model of DVT (n=8 per group) and TNF-α-induced HUVEC injury model
Comparison
HXXZ treatment vs control models
Design
Preclinical in vivo and in vitro mechanistic study
Authors
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Animal findings support mechanistic exploration of HXXZ in DVT; leaves open clinical translation pending human trials.
Does Huo-Xue-Xiao-Zhong Decoction prevent thrombus formation in preclinical models of deep vein thrombosis?
Huo-Xue-Xiao-Zhong Decoction prevents thrombus formation in DVT models by targeting SOAT2 and modulating the AKT and ERK pathways.
Li et al. (2026) studied Deep vein thrombosis. Huo-Xue-Xiao-Zhong Decoction (HXXZ) was evaluated on Thrombus length and mass. Huo-Xue-Xiao-Zhong Decoction significantly reduced thrombus length and mass in vivo and attenuated endothelial dysfunction in vitro by downregulating SOAT2 and suppressing the AKT-ERK pathway.
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