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Demonstrates anti-inflammatory effects of palmitoleic acid in cell models, suggesting therapeutic benefits.
expression. Molecular docking analysis suggested that the interaction between POA and NRF2 may involve key residues including valine (VAL-418, VAL-512 and VAL-465). Overall, POA alleviated cell inflammation through the NRF2/TBK1 signaling pathway. This study provides insights into the anti-inflammatory mechanisms of POA, highlighting its potential as a therapeutic agent.
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Sun et al. (2026) studied this question.
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