Key result
Swimming training for 8 weeks significantly reduced resting heart rate in rats (330 vs 355 bpm) and increased cardiac vagal effect (158 vs 103 bpm) compared to sedentary controls.
Why the study?
Does swimming training increase cardiac vagal activity and induce cardiac hypertrophy in male Wistar rats?
Population
24 male Wistar rats (200-220 g)
Comparison
Swimming training vs Sedentary control group
Design
Preclinical
Follow-up
8 weeks
Authors
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Should not yet change clinical exercise guidance; leaves open translation of vagal bradycardia and hypertrophy to human training models.
Does swimming training increase cardiac vagal activity and induce cardiac hypertrophy in male Wistar rats?
Absolute Event Rate: 330% vs 355%
Swimming training in rats induces resting bradycardia primarily mediated by increased parasympathetic activity, alongside physiological cardiac hypertrophy.
Medeiros et al. (2004) studied this question. Swimming training vs. Sedentary was evaluated on Resting heart rate. Swimming training for 8 weeks significantly reduced resting heart rate in rats (330 vs 355 bpm) and increased cardiac vagal effect (158 vs 103 bpm) compared to sedentary controls.
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