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October 1, 1999Arteriosclerosis Thrombosis and Vascular BiologyOpen Access

Detection, Quantification, and Characterization of Potentially Atherogenic Triglyceride-Rich Remnant Lipoproteins

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Key result

Various approaches can identify and quantify triglyceride-rich lipoprotein remnants, but more accurate and clinically applicable assays are needed to define coronary artery disease risk.

Design

Review

Authors

JCJeffrey S. CohnGeneral / Preventive / LipidsCMCaroline MarcouxGF Strong Rehabilitation CentreJDJean DavignonGeneral / Preventive / Lipids

Discussion

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Implication

Current methods insufficient for routine use; leaves open accurate CAD risk definition in hypertriglyceridemia until better assays emerge.

PICO

P
Population
Hypertriglyceridemia
E
Exposure / Comparator
Triglyceride-rich lipoprotein remnant assays

This review highlights the characteristics of triglyceride-rich lipoprotein remnants and the need for more accurate assays to define coronary artery disease risk in patients with hypertriglyceridemia.

Cite This Study

Cohn et al. (1999) conducted a review in Hypertriglyceridemia. Triglyceride-rich lipoprotein remnant assays was evaluated. Various approaches can identify and quantify triglyceride-rich lipoprotein remnants, but more accurate and clinically applicable assays are needed to define coronary artery disease risk.

synapsesocial.com/papers/6a3f7f2ca307ad3478fd44echttps://doi.org/10.1161/01.atv.19.10.2474
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Remnants of lipoproteins of intestinal and hepatic origin in familial dysbetalipoproteinemia.1983 · 106 citations
  2. 2Different patterns of postprandial lipoprotein metabolism in normal, type IIa, type III, and type IV hyperlipoproteinemic individuals. Effects of treatment with cholestyramine and gemfibrozil.1987 · 263 citations
  3. 3Relation of triglyceride metabolism and coronary artery disease. Studies in the postprandial state.1992 · 1,135 citations
  4. 4Influx in vivo of low density, intermediate density, and very low density lipoproteins into aortic intimas of genetically hyperlipidemic rabbits. Roles of plasma concentrations, extent of aortic lesion, and lipoprotein particle size as determinants.1992 · 161 citations