Key result
Three cases of hemopericardium following acute myocardial infarction were clinically diagnosed and had a favorable outcome.
Case Report (n=3)
Prompt diagnosis and drainage may yield favorable outcomes in post-MI hemopericardium; leaves open questions on incidence and anticoagulation effects.
Hemopericardium occurring in the course of acute myocardial infarction is most frequently due to rupture of the myocardium but may be caused by bleeding from granulation tissue in areas of fibrinous pericarditis.¹The use of anticoagulants has been associated with an increase in hemopericardium of both types.² Cardiac rupture is probably always fatal, and the more benign type of bleeding is presumably involved when recovery occurs. When anticoagulant therapy is a factor the signs of pericardial fluid may regress soon after the coagulation mechanism is restored to normal. There are detailed reports * of at least three cases of myocardial infarction in which pericardial tamponade occurred and was relieved by the removal of bloody fluid. This is a report of three cases in which the diagnosis was made clinically and the outcome was favorable. Case 1. —On Aug. 10, 1953, a 69-year-old man was hospitalized because of substernal pain
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L. Theodore Lawrence (1955) conducted a case report in Hemopericardium following acute myocardial infarction (n=3). Hemopericardium was evaluated on Clinical outcome. Three cases of hemopericardium following acute myocardial infarction were clinically diagnosed and had a favorable outcome.
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