Key result
Acute kidney injury after cardiac surgery was associated with a significant and sustained rise in high-sensitivity troponin T levels compared to patients without acute kidney injury.
Why the study?
Does acute kidney injury after cardiac surgery alter the clearance and diagnostic profile of high-sensitivity troponin T?
Population
359 patients undergoing cardiac surgery, mean age 55.1 ± 10.2 years. Excluded: end-stage renal disease (ESRD).
Comparison
Cardiac surgery-induced acute kidney injury vs No acute kidney injury (n=259)
Design
Cohort
Authors
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Post-cardiac surgery hsTnT interpretation warrants caution in AKI; leaves open effects on clearance and diagnostic specificity.
Observational (n=359)
No
Does acute kidney injury after cardiac surgery alter the clearance and diagnostic profile of high-sensitivity troponin T?
Effect estimate: Beta 0.47
p-value: p=0.001
Omar et al. (2017) conducted an observational in Cardiac surgery (n=359). Acute kidney injury vs. No acute kidney injury was evaluated on Association of high-sensitivity troponin T (hsTnT) with post-cardiac surgery acute kidney injury (Beta 0.47, p=0.001). Acute kidney injury after cardiac surgery was associated with a significant and sustained rise in high-sensitivity troponin T levels compared to patients without acute kidney injury.
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