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The study aims to explore the molecular mechanisms by which Panax-notoginseng inhibits cervical cancer.

July 13, 2026Bioinformatics AdvancesOpen Access

Exploring the Molecular Basis of Panax-notoginseng Mediated Inhibition of Cervical Cancer through Machine Learning, Transcriptomics Analysis, Network Pharmacology, Molecular Docking, and In-Silico Simulation Approaches

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Randomized trial investigates molecular mechanisms of Panax-notoginseng in cervical cancer, indicating potential therapeutic pathways.

The study aims to explore the molecular mechanisms by which Panax-notoginseng inhibits cervical cancer.
Utilized transcriptomic analysis to identify differentially expressed genes linked to cervical cancer.
Employed network pharmacology to examine interactions between Panax-notoginseng and cancer-related targets.
Conducted molecular docking and in-silico simulations to assess binding affinities and interactions of key compounds.
Identified 533 differentially expressed genes associated with key cancer pathways including cell cycle and p53 signaling.
Revealed 291 overlapping targets in network pharmacology analysis between Panax-notoginseng and cervical cancer genes.
Demonstrated strong binding affinities for key compounds with core therapeutic targets, showing stable interactions.

Kamal et al. (2026) studied this question.

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