Key result
Angina pectoris results from ischemic episodes that release chemicals exciting sympathetic and vagal afferent pathways, which project via the spinothalamic tract to specific thalamic and cortical areas.
Design
Review
Authors
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Advances mechanistic understanding of cardiac afferents in angina; leaves open targeted neuromodulation trials.
This review outlines the complex neural pathways, including sympathetic and vagal afferents and the spinothalamic tract, that mediate the sensation of cardiac pain (angina pectoris).
Robert D. Foreman (1999) conducted a review in Angina pectoris. Angina pectoris results from ischemic episodes that release chemicals exciting sympathetic and vagal afferent pathways, which project via the spinothalamic tract to specific thalamic and cortical areas.
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