Key result
Naoxintong administration attenuated cardiac remodeling in postmenopausal hypertensive mice by upregulating estrogen receptors and activating the Nrf2/HO-1 signaling pathway.
Why the study?
Limited treatments are available for alleviating heart remodeling in postmenopausal hypertension.
Does naoxintong ameliorate heart remodeling in a postmenopausal hypertension mouse model?
Does naoxintong ameliorate heart remodeling in a postmenopausal hypertension mouse model?
Naoxintong attenuates pathological cardiac remodeling in postmenopausal hypertension by upregulating estrogen receptors and modulating the Nrf2/HO-1 and p38α signaling pathways.
Should not change practice in postmenopausal hypertension; leaves open translation to human trials.
Limited treatments are available for alleviating heart remodeling in postmenopausal hypertension. The cardioprotective effect of naoxintong (NXT) has been widely accepted. This study aimed to explore the effects of NXT on pathological heart remodeling in a postmenopausal hypertension mouse model in vivo and H9c2 cardiomyocytes in vitro. In vivo, ovariectomy combined with chronic angiotensin II infusion was used to establish the postmenopausal hypertension animal model. NXT significantly ameliorated cardiac remodeling as indicated by a reduced ratio of heart weight/body weight and left ventricle weight/body weight, left ventricular wall thickness, diameter of cardiomyocytes, and collagen deposition in the heart. NXT also significantly increased the expression of estrogen receptors (ERs) and downregulated the expression of nicotinamide adenine dinucleotide phosphate oxidase 2 (Nox2). In vitro, NXT treatment greatly suppressed angiotensin II-induced cardiac hypertrophy, cardiac fibrosis, and excessive oxidative stress as proven by reducing the diameter of H9c2 cardiomyocytes, expression of hypertrophy and fibrosis markers, intracellular reactive oxygen species, and oxidative enzymes. Mechanistically, NXT significantly upregulated the expression of ERs, which activated the Nrf2/HO-1 signaling pathway and inhibited the phosphorylation of the p38α pathway. Collectively, the results indicated that NXT administration might attenuate cardiac remodeling through upregulating the expression of ERs, which activated the Nrf2/HO-1 signaling pathway, inhibited the phosphorylation of the p38α signaling pathway, and reduced oxidative stress.
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Cheng et al. (2022) studied Postmenopausal hypertension. Naoxintong (NXT) vs. Control was evaluated on Cardiac remodeling (heart weight/body weight ratio, left ventricle weight/body weight, left ventricular wall thickness, diameter of cardiomyocytes, and collagen deposition). Naoxintong administration attenuated cardiac remodeling in postmenopausal hypertensive mice by upregulating estrogen receptors and activating the Nrf2/HO-1 signaling pathway.
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