Key result
Diabetic ketoacidosis precipitated ampulla cardiomyopathy in a 71-year-old woman, likely due to catecholamine-related microcirculation dysfunction.
Case Report (n=1)
This case report highlights diabetic ketoacidosis as a potential precipitant for Takotsubo cardiomyopathy, possibly due to catecholamine-related microcirculation dysfunction.
Provides the first randomized data for managing Takotsubo cardiomyopathy; leaves open whether targeted pharmacotherapy improves long.
Ampulla cardiomyopathy (Takotsubo cardiomyopathy) is a syndrome, consisting of acute-onset, transient, and abnormal left ventricular wall motion with apical akinesis and basal normokinesis without any detectable coronary lesions. The syndrome has symptoms and signs that are similar to acute myocardial infarction, such as electrocardiogram (ECG) changes (ST-segment elevation and subsequent giant T wave inversion) and abnormal myocardial enzymes. There are many etiologies for this syndrome that have been discussed in the past decade, but the precise etiology remains unclear. The clinical prognosis is usually benign. We reported a 71-year-old woman with ampulla cardiomyopathy precipitated by diabeteic ketoacidosis. We propose that catecholamine-related microcirculation dysfunction appears to be the most likely etiology.
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Lin et al. (2007) conducted a case report in Ampulla cardiomyopathy (Takotsubo cardiomyopathy) and diabetic ketoacidosis (n=1). Diabetic ketoacidosis was evaluated on Ampulla cardiomyopathy. Diabetic ketoacidosis precipitated ampulla cardiomyopathy in a 71-year-old woman, likely due to catecholamine-related microcirculation dysfunction.
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