Key result
Anthracyclines induce cardiovascular toxicity through complex mechanisms including thrombosis, myocardial atrophy, and non-apoptotic programmed cell death.
Why the study?
Emerging aspects of anthracycline-induced cardiotoxicity, including thrombosis, myocardial atrophy, and non-apoptotic programmed cell death, have received less attention in recent reviews.
Design
Review
Authors
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New anthracycline cardiotoxicity pathways identified; leaves open targeted cardioprotection strategies pending clinical validation.
This review expands the understanding of anthracycline-induced cardiotoxicity beyond oxidative stress to include thrombosis, myocardial atrophy, and non-apoptotic programmed cell death.
Antoniak et al. (2022) conducted a review in Anthracycline-induced cardiovascular toxicity. Anthracyclines was evaluated. Anthracyclines induce cardiovascular toxicity through complex mechanisms including thrombosis, myocardial atrophy, and non-apoptotic programmed cell death.
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