Key result
KCNE2 is a potent regulator of membrane excitability and ion secretion that shapes the electrophysiological landscape of both excitable and non-excitable cells.
KCNE2 is a critical regulator of membrane excitability and ion secretion, with mutations linked to human cardiac arrhythmias.
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Kcne2 deletion produces murine arrhythmia; leaves open its role in human channelopathies.
Geoffrey W. Abbott (2012) conducted a review in Cardiac arrhythmia and other KCNE2-related conditions. KCNE2 was evaluated. KCNE2 is a potent regulator of membrane excitability and ion secretion that shapes the electrophysiological landscape of both excitable and non-excitable cells.
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