Key result
Neurogenic pulmonary edema likely develops from a rapid systemic sympathetic discharge and increased intracranial pressure following central nervous system injury.
This review highlights that neurogenic pulmonary edema is driven by severe sympathetic discharge and intracranial hypertension following CNS injury, emphasizing the need for specific experimental models.
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Etiopathogenesis of neurogenic pulmonary edema remains incompletely understood; leaves open mechanistic research to guide future therapies.
Neurogenic pulmonary edema is a life-threatening complication, known for almost 100 years, but its etiopathogenesis is still not completely understood. This review summarizes current knowledge about the etiology and pathophysiology of neurogenic pulmonary edema. The roles of systemic sympathetic discharge, central nervous system trigger zones, intracranial pressure, inflammation and anesthesia in the etiopathogenesis of neurogenic pulmonary edema are considered in detail. The management of the patient and experimental models of neurogenic pulmonary edema are also discussed.
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Šedý et al. (2008) conducted a review in Neurogenic pulmonary edema. Neurogenic pulmonary edema likely develops from a rapid systemic sympathetic discharge and increased intracranial pressure following central nervous system injury.
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