Key result
Leadless pacemaker implantation causes severe symptomatic tricuspid regurgitation via subvalvular apparatus interaction.
Why the study?
Leadless pacemakers can cause significant tricuspid regurgitation, but little is known about risk factors and management strategies for leadless pacemaker-induced tricuspid regurgitation.
Does leadless pacemaker implantation cause or worsen tricuspid regurgitation in patients with symptomatic bradycardia?
Case Report (n=1)
Does leadless pacemaker implantation cause or worsen tricuspid regurgitation in patients with symptomatic bradycardia?
Leadless pacemakers can cause severe, clinically significant tricuspid regurgitation via interactions with the subvalvular apparatus, emphasizing the need for comprehensive echocardiographic surveillance.
May warrant echocardiographic surveillance after leadless pacemaker implantation; leaves open the incidence and predictors of this complication.
•PPM/ICD-related TR has relevant clinical and prognostic impact.•LPs are an alternative to avoid lead-mediated complications.•Unexpectedly, LP can still cause significant degrees of TR.•TR after LP implantation likely results from subvalvular apparatus interaction.•Little is known about risk factors and management strategies for LP-induced TR.
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Cirne et al. (2021) conducted a case report in Symptomatic bradycardia and end-stage renal disease (n=1). Leadless pacemaker was evaluated on Development of severe tricuspid regurgitation. Implantation of a leadless pacemaker resulted in progressive and severe symptomatic tricuspid regurgitation due to device interaction with the subvalvular apparatus.
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