Why the study?
Left ventricular systolic dysfunction following percutaneous patent ductus arteriosus closure is a recognized phenomenon with a varied incidence reported in the literature.
What factors are associated with the development of left ventricular systolic dysfunction following percutaneous closure of patent ductus arteriosus?
What factors are associated with the development of left ventricular systolic dysfunction following percutaneous closure of patent ductus arteriosus?
Left ventricular systolic dysfunction following percutaneous PDA closure is typically transient and asymptomatic, with delayed recovery associated with increasing age, larger indexed PDA size, and pulmonary hypertension.
May warrant LV monitoring after PDA closure in select patients; leaves open predictors for prospective validation.
Transcatheter closure has become the standard treatment for patent ductus arteriosus (PDA) with excellent short- and long-term results. Left ventricular systolic dysfunction after closure is a known phenomenon rather than a complication with varied incidence documented in literature. Here, we report our experience of six cases who developed left ventricle (LV) dysfunction following percutaneous closure of PDA. Age, PDA size indexed to body surface area and pulmonary hypertension were the parameters noted to be associated with poorer LV function after closure. The LV dysfunction was transient and asymptomatic in most with delayed recovery noted with increasing age.
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Ganesh et al. (2021) studied this question.
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