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March 1, 1994Journal of Cerebral Blood Flow & MetabolismOpen Access

Nitric Oxide Synthase Inhibition and Cerebrovascular Regulation

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Authors

Costantino IadecolaUniversity of Vermont DPDale A. PelligrinoBrigham and Women's Hospital MMMichael A. MoskowitzUniversity of Iowa

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Implication

NO modulation not ready for cerebral ischemia care; leaves open its net role in regulation and ischemic damage.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Iadecola et al. (1994) studied this question.

synapsesocial.com/papers/6a6fd78c87f9210571275922 https://doi.org/10.1038/jcbfm.1994.25

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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

1Importance of Nitric Oxide Synthase Inhibition to the Attenuated Vascular Responses Induced by Topical L-Nitroarginine during Vibrissal Stimulation1994 · 110 citations
2Nitric Oxide Mediates the Neurogenic Vasodilation of Bovine Cerebral Arteries1991 · 87 citations
3NOS neurones lie near branchings of cortical arteriolae1993 · 51 citations
4Nitro-L-arginine attenuates hypercapnic cerebrovasodilation without affecting cerebral metabolism1994 · 43 citations