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March 1, 1994ViewOpen Access

Expression of the v-crk oncogene product in PC12 cells results in rapid differentiation by both nerve growth factor- and epidermal growth factor-dependent pathways.

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Authors

BHBarbara L. HempsteadCornell UniversityRBRaymond B. BirgeRutgers, The State University of New JerseyJFJ. Eduardo FajardoAlfréd Rényi Institute of Mathematics

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Cite This Study

Hempstead et al. (1994) studied this question.

synapsesocial.com/papers/6a6ffc2626a7f98052dc86a5https://doi.org/10.1128/mcb.14.3.1964
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Nerve growth factor promotes the activation of phosphatidylinositol 3-kinase and its association with the trk tyrosine kinase.1992 · 243 citations
  2. 2Paxillin: a new vinculin-binding protein present in focal adhesions.1990 · 698 citations
  3. 3Identification and characterization of a high-affinity interaction between v-Crk and tyrosine-phosphorylated paxillin in CT10-transformed fibroblasts.1993 · 255 citations
  4. 4Nerve growth factor rapidly stimulates tyrosine phosphorylation of phospholipase C-gamma 1 by a kinase activity associated with the product of the trk protooncogene.1991 · 275 citations