February 1, 2002PEDIATRICS

Pulmonary artery occlusion pressures were normal or slightly elevated (13-16 mm Hg) and stroke volume index decreased (15.3-35.7 mL/M2), indicating pulmonary edema is not directly caused by viral myocarditis.

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Population

5 consecutive infants, ages 2 to 13 months, with enterovirus 71 (EV71) infection and pulmonary edema

Design

Case_series

Follow-up

acute stage (days)

Authors

JWJing-Ming Wu JWJieh‐Neng Wang YTYu-Chien Tsai

Discussion

Member takes

Overview

Alerts clinicians to non-cardiogenic causes in EV71 edema; leaves open permeability-targeted studies.

Structured PICO

Population

5 consecutive infants, ages 2 to 13 months, with enterovirus 71 (EV71) infection and pulmonary edema

Intervention

Direct intracardiac monitoring via simultaneously implanted pulmonary arterial and central venous catheters

Outcome

Hemodynamic profiles determined every 4 hours during the acute stagesurrogate

Acute pulmonary edema in fulminant EV71 infection is likely due to increased pulmonary vascular permeability from brainstem lesions or systemic inflammation, rather than viral myocarditis.

Cite This Study

Wu et al. (2002) studied this question.

synapsesocial.com/papers/6a703402a528af2d65c362f9 https://doi.org/10.1542/peds.109.2.e26

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February 1, 2002PEDIATRICS

Pulmonary artery occlusion pressures were normal or slightly elevated (13-16 mm Hg) and stroke volume index decreased (15.3-35.7 mL/M2), indicating pulmonary edema is not directly caused by viral myocarditis.

View Full Paper

Ask AI

Bookmark

Population

5 consecutive infants, ages 2 to 13 months, with enterovirus 71 (EV71) infection and pulmonary edema

Design

Case_series

Follow-up

acute stage (days)

Authors

JWJing-Ming Wu JWJieh‐Neng Wang YTYu-Chien Tsai

Discussion

Member takes

Overview

Alerts clinicians to non-cardiogenic causes in EV71 edema; leaves open permeability-targeted studies.

Structured PICO

Population

5 consecutive infants, ages 2 to 13 months, with enterovirus 71 (EV71) infection and pulmonary edema

Intervention

Direct intracardiac monitoring via simultaneously implanted pulmonary arterial and central venous catheters

Outcome

Hemodynamic profiles determined every 4 hours during the acute stagesurrogate

Acute pulmonary edema in fulminant EV71 infection is likely due to increased pulmonary vascular permeability from brainstem lesions or systemic inflammation, rather than viral myocarditis.

Cite This Study

Wu et al. (2002) studied this question.

synapsesocial.com/papers/6a703402a528af2d65c362f9 https://doi.org/10.1542/peds.109.2.e26

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

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