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Periods of increased activity were found in the urinary bladder of the rat in situ 50–90 h after postganglionic parasympathetic denervation. The activity was not affected by hexamethoniuni, eserine produced an increase of the activity, and atropine and Hoechst 9980 totally abolished it. It is concluded that the activity is due to transmitter leaking from degenerating postganglionic nerve terminals and that the transmitter probably is acetylcholine.
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Mats Elmér (1973) studied this question.
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