Why the study?
Does inhalation of side-stream smoke or concentrated ambient fine particles accelerate atherosclerosis plaque progression in ApoE(-/-) mice?
Does inhalation of side-stream smoke or concentrated ambient fine particles accelerate atherosclerosis plaque progression in ApoE(-/-) mice?
Both environmental tobacco smoke and concentrated ambient fine particles significantly accelerate atherosclerosis progression in a mouse model, with ambient particles showing comparable effects at much lower mass concentrations.
No takes yet. Share an insight, caveat, or question.
Hypothesis-generating in ApoE(-/-) mice; leaves open translation to human cardiovascular risk and exposure guidelines.
Chen et al. (2010) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: