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February 1, 1997Antimicrobial Agents and ChemotherapyOpen Access

Transferable hyperproduction of TEM-1 beta-lactamase in Shigella flexneri due to a point mutation in the pribnow box

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Authors

LSL. Kristopher SiuNational Health Research InstitutesPHPak‐Leung HoHospital Authority
Kwok‐Yung Yuen
Kwok‐Yung YuenUniversity of Hong Kong

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Cite This Study

Siu et al. (1997) studied this question.

synapsesocial.com/papers/6a70bacd5671bbf00bc2c307https://doi.org/10.1128/aac.41.2.468
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Point mutation in the pribnow box, the molecular basis of beta-lactamase overproduction in Klebsiella oxytoca1995 · 50 citations
  2. 2A functional classification scheme for beta-lactamases and its correlation with molecular structure1995 · 2,482 citations
  3. 3<i>Escherichia coli</i>K-12 Mutants Hyperproducing Chromosomal Beta-Lactamase by Gene Repetitions1977 · 91 citations
  4. 4<i>Klebsiella oxytoca</i>: Resistance to aztreonam by overproduction of the chromosomally encoded β-lactamase1994 · 32 citations
  5. 5Activity of clavulanate combinations against TEM-1 β-lactamase-producing <i>Escherichia coli</i> isolates obtained in 1982 and 19891991 · 36 citations