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January 1, 2000Seminars in Thrombosis and Hemostasis

Asymmetric Dimethylarginine, Derangements of the Endothelial Nitric Oxide Synthase Pathway, and Cardiovascular Diseases

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Authors

RBRainer H. BögerVascular Medicine SBStefanie M. Bode‐BögerVascular Medicine

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Implication

ADMA targeting merits exploration in cardiovascular trials; leaves open whether it alters clinical outcomes.

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Cite This Study

Böger et al. (2000) studied this question.

synapsesocial.com/papers/6a70c6dcfebe604dd7098a0f https://doi.org/10.1055/s-2000-13210

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Response of cerebral blood vessels to an endogenous inhibitor of nitric oxide synthase1995 · 159 citations
2Nitric oxide-generating vasodilators and 8-bromo-cyclic guanosine monophosphate inhibit mitogenesis and proliferation of cultured rat vascular smooth muscle cells.1989 · 2,172 citations
3Impaired vasodilation of forearm resistance vessels in hypercholesterolemic humans.1990 · 1,021 citations
4Inhibition of low‐density lipoprotein oxidation by nitric oxide Potential role in atherogenesis1993 · 403 citations
5Synthesis of N<sup>G</sup>, N<sup>G</sup>Dimethylarginine by Human Endothelial Cells1993 · 81 citations