Why the study?
Does β-AR blockade reduce ER stress and apoptosis in experimental models of cardiac hypertrophy and heart failure?
Population
Sprague-Dawley rats with cardiac hypertrophy and heart failure induced by abdominal aortic constriction or…
Design
Preclinical
Follow-up
4 or 8 weeks (banding model), 2 weeks (isoproterenol model)
Authors
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May inform β-blocker mechanisms in experimental HF; leaves open translation to human therapy.
Does β-AR blockade reduce ER stress and apoptosis in experimental models of cardiac hypertrophy and heart failure?
β-AR blockade reverses ER stress and apoptosis in experimental models of heart failure, providing a mechanistic explanation for their established therapeutic benefit.
Ni et al. (2011) studied this question.
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