Key result
Folic acid and B vitamin supplementation do not lower cardiovascular event risk, and measuring homocysteine levels has not proven useful for guiding treatment.
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Although evidence suggests that the homocysteine hypothesis is still relevant as a predictor of cardiovascular risk, we cannot conclude that measuring the homocysteine level is useful in guiding treatment. Furthermore, studies of primary and secondary prevention show no evidence that taking folic acid or other B vitamins lowers the risk of cardiovascular events.
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Abraham et al. (2010) studied this question. Folic acid and B vitamin supplementation do not lower cardiovascular event risk, and measuring homocysteine levels has not proven useful for guiding treatment.
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