Population
Rats with acute renal failure induced by glycerol, subdivided into oliguric, nonoliguric, and polyuric groups
Design
Preclinical
Authors
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Hypothesis-generating for urinary ACE as proximal tubule injury marker in experimental AKI; human validation required before clinical consideration.
In a rat model of glycerol-induced acute renal failure, increased urinary ACE activity is associated with proximal tubule damage, suggesting its potential as a biomarker for tubular injury.
Pedraza‐Chaverrí et al. (1995) studied this question.
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