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January 1, 2006Journal of Pharmacological SciencesOpen Access

Blockade by NIP-142, an Antiarrhythmic Agent, of Carbachol-Induced Atrial Action Potential Shortening and GIRK1/4 Channel

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Authors

TMTomoyuki MatsudaChiba University MIMie ItoToho University SISayoko Ishimaru

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Implication

NIP-142's tertiapin-mimicking atrial effects remain in vitro; leaves open development of IK,ACh-targeted antiarrhythmics.

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Cite This Study

Matsuda et al. (2006) studied this question.

synapsesocial.com/papers/6a73dc63e3ef2a9a940dd748 https://doi.org/10.1254/jphs.fp0060324

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Action Potential Shortening and Negative Inotropic Effects of a Novel Potassium Channel Opener, NIP-121, as Compared with Cromakalim in Guinea Pig Ventricular Myocardium1991 · 8 citations
2Effect of Cilnidipine on L- and T-type Calcium Currents in Guinea Pig Ventricle and Action Potential in Rabbit Sinoatrial Node2004 · 17 citations
3Evaluation of the role of IKAChin atrial fibrillation using a mouse knockout model2001 · 242 citations
4Regulation of spontaneous opening of muscarinic K+ channels in rabbit atrium.1991 · 85 citations
5Lack of Action Potential-Prolonging Effect of Terfenadine on Rabbit Myocardial Tissue Preparations2004 · 26 citations