Theoretical hypothesis proposes that energetic overload influences early cancer development, suggesting new avenues for research.
The earliest stages of carcinogenesis remain incompletely understood despite extensive knowledge of genetic and molecular alterations associated with malignant transformation. This paper proposes a complementary biophysical hypothesis suggesting that persistent energetic overload associated with membrane homeostasis may contribute to early cellular adaptation before detectable genetic changes become dominant.
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Peter Mikuláš (2026) studied this question.
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