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May 25, 2005Cardiovascular Research

Sodium channels as macromolecular complexes: Implications for inherited arrhythmia syndromes

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Authors

LMLaurence S. MeadowsUniversity of Michigan LILori L. IsomElectrophysiology

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Implication

May support targeted therapies in inherited arrhythmias; leaves open clinical translation pending validation studies.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Meadows et al. (2005) studied this question.

synapsesocial.com/papers/6a7620acf1e99ff85ab23b3e https://doi.org/10.1016/j.cardiores.2005.04.003

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Slowed conduction and ventricular tachycardia after targeted disruption of the cardiac sodium channel gene <i>Scn5a</i>2002 · 391 citations
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5Dystonia associated with mutation of the neuronal sodium channel Scn8a and identification of the modifier locus Scnm1 on mouse chromosome 31999 · 72 citations