Key result
This document is a journal masthead and contains no clinical study data.
Why the study?
Right ventricular failure is a critical cause of morbidity and mortality in acute pulmonary embolism, and a subset of patients develop chronic RV dysfunction with or without pulmonary hypertension.
This review highlights the pathophysiology, risk stratification, and management of right ventricular dysfunction following acute pulmonary embolism.
RV dysfunction refines acute PE risk stratification; leaves optimal chronic management strategies open.
Right ventricular (RV) failure is a critical cause of morbidity and mortality in patients presenting with pulmonary embolism (PE). The presentation of RV failure is based on the combination of clinical findings, laboratory abnormalities, and imaging evidence. An improved understanding of the pathophysiology of RV dysfunction following PE has given rise to more accurate risk stratification and broader therapeutic approaches. A subset of patients with PE develop chronic RV dysfunction with or without pulmonary hypertension. In this review, we focus on the impact of PE on the RV and its implications for risk stratification, prognosis, acute management, and long-term therapy.
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Bejjani et al. (2022) studied this question. This document is a journal masthead and contains no clinical study data.
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