Why the study?
Traditional Chinese medicine offers advantages in treating early or intermediate heart failure, but the in vivo therapeutic efficacy and molecular targets of Xin-shu-bao across different stages of post-infarction heart failure required investigation.
Does Xin-shu-bao (XSB) improve cardiac function and reduce fibrosis in a mouse model of HFrEF following myocardial infarction?
Does Xin-shu-bao (XSB) improve cardiac function and reduce fibrosis in a mouse model of HFrEF following myocardial infarction?
Xin-shu-bao demonstrates cardioprotective efficacy in a mouse model of HFrEF post-MI by improving cardiac function and reducing fibrosis via modulation of THBD/ARRB1/FGF1/STIM1 signaling.
XSB efficacy across HF stages in mice leaves open human translation and target validation.
Traditional Chinese medicine offer unique advantages in mitigating and preventing early or intermediate stage for treating heart failure (HF). The purpose of this study was to assess the in vivo therapeutic efficacy of Xin-shu-bao (XSB) at different stages of HF following induction of a myocardial infarction (MI) in mice and use mass spectrometry-based proteomics to identify potential therapeutic targets for different stages of HF based on the molecular changes following XSB treatment. XSB had high cardioprotective efficacy in the pre-HF with reduced ejection fraction (HFrEF) stages, but had a weak or no effect in the post-HFrEF stages. This was supported by echocardiographic measurements showing that XSB decreased ejection fraction and fractional shortening in HF. XSB administration improved cardiac function in the pre- and post-HFrEF mouse model, ameliorated deleterious changes to the morphology and subcellular structure of cardiomyocytes, and reduced cardiac fibrosis. Proteomics analysis showed that XSB intervention exclusively targeted thrombomodulin (THBD) and stromal interaction molecule 1 (STIM1) proteins when administered to the mice for both 8 and 6 weeks. Furthermore, XSB intervention for 8, 6, and 4 weeks after MI induction increased the expression of fibroblast growth factor 1 (FGF1) and decreased arrestin β1 (ARRB1), which are classic biomarkers of cardiac fibroblast transformation and collagen synthesis, respectively. Overall, the study suggests that early intervention with XSB could be an effective strategy for preventing HFrEF and highlights potential therapeutic targets for further investigation into HFrEF remediation strategies.
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Zhang et al. (2023) studied this question.
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