Residue 135 of Tropomodulin-1 is critical for its interaction with tropomyosin, proper subcellular localization, and maintaining sarcomere integrity in cardiomyocytes.
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Supports residue 135 as critical for Tmod-1 sarcomere function; leaves open relevance to human cardiomyopathy.
Kong et al. (2006) studied this question.
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