Role of adiponectin in diabetes myocardial ischemia-reperfusion injury and ischemic postconditioning

Figures

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  Figure 1. The effect of IPO on myocardial infarcted area on N and DM rats subjected to IR in the presence and absence of IPO. The blue-stained areas represent non-ischemic tissue, red-stained areas represent the area at risk (AAR), and pale areas represent infarct areas (IA). Values are expressed as mean ± SD (n=8). * P < 0.05, # P < 0.05 vs . NIR group.

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  Figure 2. - The level of MDA and Nitrotyrosine in myocardium and serum level of 15-F2t-isoprostane in N and DM rats subjected to IR in the presence and absence of IPO. Values are expressed as mean ± SD (n=8). * P < 0.05 vs . NS group, # P < 0.05 vs . NIR group.

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  Figure 3. - The change of cardiac function on N and DM rats subjected to IR in the presence and absence of IPO. Values are expressed as mean ± SD (n=8). * P < 0.05 vs . NS group, # P < 0.05, & P < 0.05 vs . NIR group.

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  Figure 4. The level of plasma adiponectin in N and DM rats subjected to IR in the presence and absence of IPO. Values are expressed as mean ± SD (n=8). * P < 0.05 vs . NS group, # P < 0.05 vs . NIR group.

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  Figure 5. - The change of p-Akt and total-Akt expression in the presence and absence of IPO. Values are expressed as mean ± SD (n=8). * P < 0.05 vs . NS group, # P < 0.05 vs . NIR group.

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  Figure 6. The relationship between ANP and myocardial infarcted area, P-Akt. The simple linear regression between plasma adiponectin levels and myocardial infarct size together with p-Akt/ Akt protein expression in N and DM rats subjected to IR, with or without IR.