Why the study?
Does overexpression of a dominant negative Gsα-mutant protect against isoproterenol-induced hypertrophy and alter myocardial function in mice?
Does overexpression of a dominant negative Gsα-mutant protect against isoproterenol-induced hypertrophy and alter myocardial function in mice?
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Attenuating Gsα-dependent signaling protects against isoproterenol-induced cardiac hypertrophy in a transgenic mouse model, offering insights into β-adrenergic modulation in heart failure.
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Streit et al. (2016) studied this question.
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