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January 26, 2004American Journal of Respiratory Cell and Molecular Biology

The Splicing and Fate of ADAM33 Transcripts in Primary Human Airways Fibroblasts

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Authors

RPRobert M. PowellSouthampton General HospitalJWJames WicksSouthampton General HospitalJHJohn W. HollowayUniversity of Southampton

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Implication

May guide isoform-selective asthma research; leaves open clinical relevance pending functional validation.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Powell et al. (2004) studied this question.

synapsesocial.com/papers/6a7d545e149bd8e2c5749d98https://doi.org/10.1165/rcmb.2003-0330oc
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Also Consider

Synapse has enriched 3 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1The ADAMs family of metalloproteases: multidomain proteins with multiple functions2003 · 1,065 citations
  2. 2Roles of Meltrin β/ADAM19 in the Processing of Neuregulin2001 · 181 citations
  3. 3ADAM 33: just another asthma gene or a breakthrough in understanding the origins of bronchial hyperresponsiveness?2003 · 24 citations