Population
Rodent models of salt-sensitive hypertension
Design
Review
Authors
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Does not support practice changes in hypertension; leaves open translation of rodent pathways to human therapy.
This review elucidates novel molecular pathways involving mineralocorticoid and glucocorticoid receptors that contribute to impaired renal excretory function and salt-sensitive hypertension, highlighting potential therapeutic targets.
Toshiro Fujita (2014) studied this question.
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