Idiopathic left ventricular tachycardia localised at the distal end of the posterior fascicle by non-contact activation mapping

Lars Eckardt; G Breithardt; Wilhelm Haverkamp

doi:10.1136/heart.87.4.374

April 1, 2002HeartOpen Access

Idiopathic left ventricular tachycardia localised at the distal end of the posterior fascicle by non-contact activation mapping

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Why the study?

Does permanent inhibition of guanylyl cyclase A receptor affect cardiac function and structure in mice?

Structured PICO

Does permanent inhibition of guanylyl cyclase A receptor affect cardiac function and structure in mice?

P

Population

GC-A-/- and corresponding wild type mice (GC-A+/+)

I

Intervention

Permanent inhibition of guanylyl cyclase A receptor (GC-A knockout)

C

Comparator

Wild type mice (GC-A+/+)

O

Outcome

Cardiac function and histological characteristicssurrogate

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Implication

Permanent inhibition of the guanylyl cyclase A receptor in mice leads to progressive cardiac changes, starting with compensated hypertrophy and advancing to interstitial fibrosis and impaired contractility.

Abstract

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Discussion

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Cite This Study

Eckardt et al. (2002) studied this question.

synapsesocial.com/papers/6a7e3725d67092c043990a09 https://doi.org/10.1136/heart.87.4.374

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Idiopathic left ventricular tachycardia localised at the distal end of the posterior fascicle by non-contact activation mapping

Structured PICO

Ask about this paper

Implication

Abstract

Discussion

Cite This Study

Idiopathic left ventricular tachycardia localised at the distal end of the posterior fascicle by non-contact activation mapping

Structured PICO

Ask about this paper

Implication

Abstract

Discussion

Cite This Study

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