This canine model of chronic myocardial infarction demonstrates electrophysiologic and histopathologic heterogeneity that makes it highly susceptible to pacing-induced ventricular tachyarrhythmias, providing a useful model for studying arrhythmia mechanisms.
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Validates chronic MI canine model for VT studies; leaves open whether small infarcts confer similar risk in patients.
Michelson et al. (1981) studied this question.
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