The interaction of the Kv1.1 channel C-terminus with the cytoskeleton, along with its phosphorylation state, regulates the extent of inactivation conferred by the beta subunit.
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Supports cytoskeletal-phosphorylation interplay in Kv1.1 inactivation in vitro; leaves open translation to cardiac excitability or arrhythmias.
Jie et al. (1997) studied this question.
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