Key result
Left ventricular wall motion abnormalities may directly increase stroke risk through thrombus formation or merely indicate the burden of systemic atherosclerosis.
Left ventricular wall motion abnormalities may directly increase stroke risk or indicate systemic atherosclerosis burden, though their exact role remains unclear.
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Wall motion abnormalities should not alter anticoagulation decisions now; leaves open whether they confer independent embolic risk beyond atherosclerosis.
Left ventricular wall motion abnormalities (LVWMAs) are commonly observed in a variety of medical conditions, including coronary artery disease, congestive heart failure, stress-induced cardiomyopathy, myocarditis, chronic renal disease, and stroke.1–4 Their underlying disease mechanisms and their potential causative role for stroke remain inadequately elucidated. LVWMAs may directly increase stroke risk through thrombus formation in the left ventricle5 or may merely indicate the burden of systemic atherosclerosis underlying stroke, and not the primum movens.
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Raf Brouns (2017) conducted a review in Left ventricular wall motion abnormalities. Left ventricular wall motion abnormalities was evaluated. Left ventricular wall motion abnormalities may directly increase stroke risk through thrombus formation or merely indicate the burden of systemic atherosclerosis.
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