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Unresolved questions on inflammation and infection in ACS pathogenesis persist; leaves open the need for mechanistic and therapeutic trials.
Extensive evidence supports a pathogenic role for both local and systemic inflammation in acute coronary syndromes. However, several important questions remain unanswered. Is the observed inflammatory process a precursor or a consequence of coronary plaque rupture? Is the inflammatory component of unstable coronary disease a potential therapeutic target? Finally, do infectious agents have a pathogenic role in coronary atherosclerosis and acute coronary syndromes?
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Niall Mulvihill (2002) studied this question.
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