Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
November 1, 1995Molecular and Cellular BiologyOpen Access

Genetic Enhancement of RNA-Processing Defects by a Dominant Mutation in B52, the Drosophila Gene for an SR Protein Splicing Factor

View Full Paper
Ask AI
Bookmark
Share

Authors

XPXianbu PengColumbia UniversitySMStephen M. MountUniversity of Maryland, College Park

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Peng et al. (1995) studied this question.

synapsesocial.com/papers/6a81cf141ece0ba82ce1b565https://doi.org/10.1128/mcb.15.11.6273
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1The SR protein B52/SRp55 is essential for Drosophila development.1994 · 112 citations
  2. 2Synergistic interactions between two distinct elements of a regulated splicing enhancer.1995 · 156 citations
  3. 3SR Proteins Promote the First Specific Recognition of Pre-mRNA and Are Present Together with the U1 Small Nuclear Ribonucleoprotein Particle in a General Splicing Enhancer Complex1994 · 46 citations
  4. 4General splicing factor SF2/ASF promotes alternative splicing by binding to an exonic splicing enhancer.1993 · 288 citations
  5. 5Resonance Assignments and Solution Structure of the Second RNA-Binding Domain of Sex-lethal Determined by Multidimensional Heteronuclear Magnetic Resonance1994 · 50 citations