Why the study?
How do seated and supine passive recovery following supramaximal exercise affect stroke index and systemic vascular resistance index in healthy men?
How do seated and supine passive recovery following supramaximal exercise affect stroke index and systemic vascular resistance index in healthy men?
Stroke index persistently rises during the first 10 minutes of passive seated recovery following supramaximal exercise, likely due to sustained attenuation in systemic vascular resistance index.
Answers are grounded in this paper and cite their sources.
No immediate clinical implications for recovery protocols; leaves open posture effects on postexercise hemodynamics in patients.
The cardiodynamic responses of 9 healthy men (mean age +/- SD, 22.3 +/- 2.0 yrs) were measured during seated and supine passive recovery following the Wingate Anaerobic Power Test (WAPT). Stroke index (SI) was determined noninvasively using impedance cardiographic techniques. During the initial stages of seated recovery, SI progressively increased (1 min, 60.4 +/- 6.6 ml/m2; 3 min, 64.6 +/- 5.9 ml/m2) and achieved peak levels by 5 min (70.5 +/- 6.2 ml/m2). Between Minutes 3 and 10 of seated recovery, SI was significantly (p < or = 0.05) higher than the preexercise value (46.0 +/- 4.0 ml/m2). A similar response pattern for SI was observed during supine recovery. The systemic vascular resistance index (SVRI) decreased by 101% and 114% from preexercise baseline values after 1 min of recovery in the supine and seated postures, respectively. The persistent rise in SI during the first 10 min of passive seated recovery may be explained by the sustained attenuation in SVRI coupled with the anticipated residual myocardial inotropic effects following the WAPT.
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Kilgour et al. (1995) studied this question.
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