Why the study?
Cardiomyocyte apoptosis is an important factor in cardiac function decline, but progress and cooperative relationships in this research field needed to be understood.
This bibliometric analysis identifies key research trends and frontiers in cardiomyocyte apoptosis, highlighting the NLRP3 inflammasome, circular RNA, and sepsis as emerging areas of focus.
Steers research funding toward NLRP3, circular RNA, and sepsis in cardiomyocyte apoptosis; leaves clinical translation open.
Cardiomyocyte apoptosis is an important factor in cardiac function decline observed in various cardiovascular diseases. To understand the progress in the field of cardiomyocyte apoptosis research, this paper uses bibliometrics to statistically analyze publications in this field. A total of 5939 articles were retrieved from the core Web of Science database, and then VOSviewer and Citespace were used to conduct a scientometric analysis of the authors, countries, institutions, references and keywords included in the articles to determine the cooperative relationships between researchers that study cardiomyocyte apoptosis. At present, the research hotspots in this field mainly include experimental research, molecular mechanisms, pathophysiology and cardiac regeneration of cardiomyocyte apoptosis-related diseases. NOD-like receptor thermal protein domain associated protein 3 inflammasome, circular RNA, and sepsis are the research frontiers in this field and are emerging as new areas of research focus. This work provides insight into research directions and the clinical application value for the continued advancement of cardiomyocyte apoptosis research.
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Wang et al. (2023) studied this question.
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