Why the study?
Patients with pulmonary hypertension typically present with a diminished platelet count, but the role of platelets in PH development and progression remains unclear.
Population
Animal models of PH and patients with PH
Comparison
Platelet depletion, healthy vs PH platelet transfusion, and pharmacological targeting of ICAM-1
Design
Preclinical animal and translational mechanistic study
Authors
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ICAM-1 inhibition may attenuate hypoxic pulmonary hypertension; leaves open translation to human disease.
Platelet-endothelial adhesion mediated by glycoprotein IIb/IIIa and ICAM-1 exacerbates pulmonary hypertension by intensifying pulmonary vascular wall thickening, highlighting a potential mechanistic therapeutic target.
Chen et al. (2025) studied this question.
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