Why the study?
Does MCI-154 alter membrane potentials and currents in isolated guinea pig ventricular cells?
Does MCI-154 alter membrane potentials and currents in isolated guinea pig ventricular cells?
The positive inotropic action of the novel cardiotonic drug MCI-154 may be partially mediated by a decrease in the inward rectifier K+ current and increased Ca2+ sensitivity.
No immediate clinical implications for MCI-154; leaves open translation of IK1 effects to intact hearts or patients.
Effects of MCI-154, a newly synthesized cardiotonic drug with vasodilator property, on the membrane potential and currents of the isolated papillary muscles and the single ventricular cells of the guinea pig were examined. MCI-154 (10(-7)-10(-4) M) increased the developed tension in a dose-dependent manner without significant changes in the action potential configuration of the guinea pig papillary muscle. The slow response action potential obtained by increasing the extracellular K+ concentration to 24 mM was augmented by MCI-154 (10(-6) and 10(-4) M). This effect of MCI-154 was not blocked by atenolol (10(-6) M). In voltage clamp experiments on single ventricular cells, the slow inward current was not affected, but the inward rectifier K+ current was reduced by higher concentration of MCI-154 (10(-4) M). Considering the "run down" phenomenon of the slow inward current, the possibility that MCI-154 might have a slight increasing effect on the slow inward current could not be ruled out. From these results, it can be concluded that the positive inotropic action of MCI-154 can be explained partially by the decrease in the inward rectifier K+ current, and probably by the increase in the Ca2+ sensitivity of the contractile protein system.
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KATAYAMA et al. (1987) studied this question.
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