The interaction of Lp(a) with cells is distinct from its interaction with extracellular matrix and plasmin-treated fibrinogen, indicating that the recognition sites within Lp(a) and plasminogen for these regulatory molecules are not identical.
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Distinguishes Lp(a) cell binding from matrix interactions; hypothesis-generating for domain-specific antagonists in atherothrombosis.
A 1999 study studied this question.
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