Intravenous verapamil, commonly used for PSVT termination, can rarely induce polymorphic ventricular tachycardia.
IV verapamil may rarely provoke polymorphic VT in PSVT; case reports leave incidence and risk factors undetermined.
Verapamil is widely used for the termination of paroxysmal supraventricular tachycardia (PSVT) with little proarrhythmic effect. We describe two cases of PSVT that changed to non-sustained polymorphic ventricular tachycardia after administration of verapamil. Electrophysiological study revealed atrioventricular nodal reentrant tachycardia in the first case, and atrioventricular reentrant tachycardia due to a concealed left lateral accessory pathway in the second case. Catecholamine-induced automaticity was one of the possible mechanisms of VT in the first case, but the mechanism is unknown in the second case.
No takes yet. Share an insight, caveat, or question.
Shiraishi et al. (2002) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: