In a mouse model of excessive erythrocytosis, decreased hypercapnic ventilatory response is driven by reduced metabolism rather than altered central chemosensitivity by erythropoietin.
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Findings in Tg6 mice warrant caution extrapolating to humans; leaves open Epo effects on ventilatory control in clinical erythrocytosis.
Laouafa et al. (2016) studied this question.
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