Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
June 29, 2023European Heart Journal

Culprit plaque morphology determines inflammatory risk and clinical outcomes in acute coronary syndrome

View Full Paper
Ask AI
Bookmark
Share

Why the study?

It was uncertain whether clinical outcomes differ following RFC-ACS versus IFC-ACS and whether outcomes are affected by a specific inflammatory response.

Does culprit plaque morphology (intact vs. ruptured fibrous cap) determine inflammatory risk and clinical outcomes in patients with acute coronary syndrome?

Population

398 consecutive ACS patients

Comparison

RFC-ACS vs IFC-ACS

Design

Prospective translational cohort study

Follow-up

2 years

Authors

TGTeresa GerhardtCSClaudio SeppeltYAYoussef S. Abdelwahed

Discussion

Loading...

Member takes

Overview

Supports lower MACE+ with IFC-ACS; leaves open whether plaque morphology should guide risk stratification in ACS.

Structured PICO

Does culprit plaque morphology (intact vs. ruptured fibrous cap) determine inflammatory risk and clinical outcomes in patients with acute coronary syndrome?

P
Population
398 consecutive patients with acute coronary syndrome (ACS)
I
Intervention
Intact fibrous cap (IFC) culprit lesion phenotype
C
Comparator
Rupture of the fibrous cap (RFC) culprit lesion phenotype
O
Outcome
Composite of cardiac death, recurrent ACS, hospitalization for unstable angina, and target vessel revascularization at 2 years [major adverse cardiovascular events (MACE+)]composite

In patients with acute coronary syndrome, an intact fibrous cap culprit lesion is associated with a distinct, lower inflammatory response and a significantly lower 2-year risk of major adverse cardiovascular events compared to plaque rupture.

Cite This Study

Gerhardt et al. (2023) studied this question.

synapsesocial.com/papers/6a83a6d76de9cd8ef3ee74a2https://doi.org/10.1093/eurheartj/ehad334
View Full Paper
Ask AI
Bookmark
Share